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BIOTIN AND PARKINSON’S: A NEW FRONTIER IN NEUROSCIENCE A breakthrough study has uncovered a potential link between biotin (vitamin B7) metabolism and Parkinson’s disease, offering a new avenue for intervention. Researchers found that manganese exposure disrupts biotin levels, leading to neurotoxicity. But biotin supplementation reversed these effects—even in human neurons. Manganese exposure reduced bioavailable biotin, triggering neurodegeneration. Drosophila (fruit fly) models showed Parkinson-like symptoms, which biotin alleviated. Human neurons responded similarly, with biotin protecting against cellular damage. This could pave the way for biotin-based therapies for Parkinson’s patients. More research is needed, but the results are promising. Source: Science Signaling